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Showing posts with label Dr. William R. Davis. Show all posts
Showing posts with label Dr. William R. Davis. Show all posts

Sunday, January 5, 2014

Published on Dec 30, 2013
          
Heart disease prevention and reversal pioneers Dr. William Davis and Dr. William Blanchet meeting for the first time to have a frank discussion on how to beat heart disease and heat attack. This is the first of a multi-part series documenting this historic meeting.

http://www.youtube.com/watch?v=hYiZtfNBI80

Wednesday, September 18, 2013

Thyroid Pills & Heart Disease - Dach

Thyroid Pills Prevent Heart Attacks by Jeffrey Dach MD


bottle,pills,thyroid,westhroid,capsulesThyroid Pills Prevent Heart Attacks by Jeffrey Dach MD

The Low Thyroid Condition and Heart Disease

In 1976 Broda Barnes was the first to connect low thyroid function with heart attacks and heart disease.  His book is called, Hypothyroidism The Unsuspected Illness by Broda Barnes, Click Here to read my Book Review..

Above Left Image: Photo of WP Thyroid Pills Bottle courtesy of RLC labs Natural thyroid pills

Discovering the Connection
How did Broda Barnes discover the connection between low thyroid and heart disease?  Barnes took summer vacations in Graz Austria every year to study the autopsy files.  Graz had a high prevalence of thyroid disorders, and anyone in Graz who died over the past 100 years required an autopsy to determine cause of death, as mandated by the authorities.  This rather large amount of autopsy data showed that low thyroid patients survived the usual childhood infectious diseases thanks to the invention of antibiotics, and years later develop heart disease.  Barnes also found that thyroid treatment was protective in preventing heart attacks, based on his own clinical experience.  Likewise for diabetes, Dr. Barnes found that adding thyroid medication was beneficial at preventing the onset of vascular disease in diabetics.  Again, blood tests are usually normal.
New research like the Hunt Study confirms that Broda Barnes was right all along, creating a paradigm shift in thyroid treatment, and constituting a frontal assault on the Institution of Medicine’s thyroid dogma.

The Hunt Study - Thyroid Function and  Heart Disease
 
TSH is short for thyroid stimulating hormone, made by the pituitary gland.  TSH actually stimulates the thyroid gland to make more thyroid hormone, and can therefore be used as a barometer of thyroid function.  If thyroid function is low, the pituitary sends out more TSH to stimulate the thyroid to make more thyroid hormone.  
Mainstream Medicine regards the TSH as the single most important test for determining thyroid function. High TSH means low thyroid function, and a Low TSH means normal or high thyroid function.


What Did The Hunt Study Find?
Thyroid GlandThe Hunt Study from the April 2008 Archives of Internal Medicine examined mortality from coronary heart disease (CHD) and TSH level.  The authors conclude,
“The results indicate that relatively low but clinically normal thyroid function may increase the risk of fatal CHD.”
Above Image: Thyroid Gland makes thyroid hormone.

The Hunt Study measured thyroid function with the TSH test in 17,000 women and 8,000 men with no known thyroid disease or heart disease. All patients had “normal TSH” levels meaning the TSH values were in the lab reference range of 0.5 to 3.5.   The women were stratified into three groups, lower TSH, intermediate and upper TSH levels, and mortality from heart disease was recorded over an 8 year observation period.

 (see chart below).
70% Increase in Heart Disease Mortality for TSH in Upper Normal Range
Heart Attack with Occluded Artery The Hunt study found that group with the higher TSH had a 70% increased mortality from heart disease compared to the lower TSH group.  Remember all these TSH vales were in the normal lab range. See chart below for results of the Hunt Study:

Left Image: Heart with Occluded Coronary Artery and Infarction at the apex, courtesy of wikimedia commons.

Results of the Hunt Study:

   TSH   Death from Heart Disease
 Group 1 0.50-1.4 baseline risk
 Group 21.5-2.440% higher than baseline
 Group 32.5-3.570% higher than baseline

 
This Finding is Earthshaking !!

This means that merely by taking natural thyroid pills to reduce TSH to the low end of “normal” (0.5), one can reduce death from cardiovascular disease by 70 percent.  This mortality benefit is mind boggling and far exceeds any drug intervention available.

Thyroid Hormone Also Improves LDL Lipo-Proteins
Another report from the Hunt Study published in 2007 showed that LDL cholesterol was linearly associated with TSH level. (see chart below).
Below Chart showing LDL cholesterol (non-HDL) goes up as TSH goes up.  Note that TSH always remains within in the lab “normal range” (0.5-3.5).
Above chart shows linear increase in LDL cholesterol as TSH increases.
Image Courtesy of Hunt Study European Journal of Endocrinology, Vol 156, Issue 2, 181-186, 2007
The Conclusion is Clear:
The best way to normalize lipoprotein profile and reduce mortality from heart disease is to reduce TSH to the lower end of the normal range with thyroid medication.  A TSH in the upper end of the normal range is associated with increased cardiovascular mortality and elevations in LDL lipo-protein measurements.  A TSH at the lower end of the normal range is associated with protection from heart disease.

Statin Drugs or Thyroid to Prevent Heart Disease in Women?
Atorvastatin
My previous article discussed the issue of statin drugs for women.  Decades of published statin drug studies show that statin drugs simply don’t work for women, and don’t reduce mortality from heart disease in women.  But on the other hand, the HUNT study shows that TSH levels in the lower normal range provide a 70% reduction in heart disease mortality for women.  This can be accomplished safely with inexpensive thyroid medication under a physician’s supervision.  So for women concerned about preventing heart disease, this is good news, pointing out a natural alternative to statin drugs that works much better.

Natural Thyroid is Better
Thyroid HormoneRather than Synthroid, we prefer to use natural thyroid which is a dessicated porcine thyroid gland from RLC Labs.  The reason for this is that we have seen better clinical results with the natural thyroid preparations compared to synthroid.
Above image: thyroid hormone Courtesy Wikimedia .
Natural Thyroid is Safer, but can Cause Adverse Effects of Palpitations
Although natural thyroid is safe, there is always the possibility of adverse effects from thyroid excess, defined as too much thyroid medication.  The first sign of thyroid excess is usually a rapid heart rate at rest or perhaps palpitations (at rest).  We spend about five minutes at the office going over this adverse effect before starting patients on thyroid medication.  Usually patients will notice the heart rate going up or the heart beat sounding louder than usual as the first sign that can be easily recognized.  Once recognized, the patient is instructed to stop the thyroid medication, and symptoms usually resolve within 6 hours (for natural thyroid).  It is perfectly safe to stop the thyroid medication at any time, as there will be no acute changes, merely a gradual reversion to the original state that existed before starting the thyroid pills.

Some patients are very sensitive to thyroid medication and will have thyroid excess symptoms such as rapid heart rate and palpitations from small amounts of thyroid medication.  These are usually the elderly with underlying heart disease and/or magnesium deficiency, and we usually avoid giving thyroid medication to these patients.  We also liberally supplement everyone with magnesium if their RBC magnesium levels are low.

About 5 per cent of our patients initially started on thyroid will notice symptoms of thyroid excess with a rapid heart rate, and they will stop the medication for a day or two and restart at a lower dosage with no problem.  This is more common in Hashimoto’s patients whose own production of thyroid hormones may fluctuate from month to month.  Patients with magnesium deficiency or adrenal fatigue with low cortisol output on salivary testing will also tend to be more sensitive to small amounts of thyroid medication, so caution is advised in these groups as well.

Thyroid Excess Can Rarely Cause Atrial Fibrillation
EKG
Upper Image: EKG strip with Red Arrow shows atrial fibrillation, no recognizable P-wave.  Lower Image: EKG strip with normal rhythm with recognizable P wave (blue arrow). Courtesy of Wikimedia Commons.
So far, we have not had a patient go into atrial fibrillation from thyroid medication, probably because we spend so much time with each patient discussing the symptoms of thyroid excess, and the importance of stopping the thyroid medication if these symptoms are noted.

Mainstream Docs Don’t Have Time To Discuss Adverse Effects
Doctor thinking about thyroid dosageOne of the reasons the mainstream conventional docs will give only a minuscule amount of synthroid to the low thyroid patient is that they simply don’t have the time to discuss thyroid excess and can’t afford an adverse event which is more likely if the patient doesn’t have a clue about what to watch out for.  In addition, mainstream medical docs don’t recognize the syndrome of adrenal fatigue or magnesium deficiency , so they can run into problems with thyroid excess without understanding why, and this also makes them very cautious, tending to under treat.
Left Image: Doctor thinking about thyroid dosage.
In patients with underlying heart disease who are prone to cardiac arrhythmias, thyroid excess can cause atrial fibrillation with characteristic EKG appearance.  Atrial fibrillation can be a problem, because if it becomes chronic and doesn’t go away on its own, the cardiologist will try a maneuver called cardioversion, the application of an electrical shock to restart a normal cardiac rhythm.  Or, if that doesn’t work, prescribe blood thinners, all of which is not without risk.  So it is better to avoid atrial fibrillation altogether by simply stopping the thyroid pills whenever symptoms of rapid heart rate or palpitations are noted while at rest.  Exercise induced rapid heart rate, of course, doesn’t count since that is normal cardiovascular response to exercise.

How To Design A Better Hunt Study
How would I design an even better Hunt Study? That’s easy. Include another group of patients with TSH levels above and below the study group, namely, below 0.5, and above 3.5.  I would also include data on annual CAT coronary calcium scores.  I would predict that the lower TSH group (below 0.5) would have even less heart disease than the higher TSH group, and that coronary calcium score, indicating plaque burden, would go up as TSH went up.

Read More About the Hunt Study from William Davis MD and Jacob Teitelbaum MD:
Is normal TSH too high?   by William Davis MD
Low Thyroid (Even if Tests are Normal) is a Major Cause of Heart Attacks by Jacob Teitelbaum, MD.

Credit and Thanks is given to William Davis MD and Jacob Teitelbaum MD for bringing the Hunt Study to my attention.

For more information on thyroid and heart disease, read my previous articles:
Saving Time Russert and George Carlin
Healthy Men Should Not Take Statin Drugs
Heart Disease Vitamin C and Linus Pauling
Getting Off Statin Drug Stories
How to Reverse Heart Disease with the Coronary Calcium Score (part one)
Reversing Heart Disease Part Three
Cholesterol Lowering Drugs for the Elderly, Bad Idea
Cholesterol Lowering Statin Drugs for Women Just Say No

Jeffrey Dach MD
7450 Griffin Road Suite 190
Davie Florida 33314
954-792-4663
www.jeffreydach.com
www.drdach.com
www.naturalmedicine101.com
www.truemedmd.com
(c) 20013 Jeffrey Dach MD All Rights Reserved, This article may be reproduced on the internet without permission, provided there is a link to this page and proper credit is given.
Link to this article:http://wp.me/P3gFbV-LU
http://jeffreydach.com/2008/10/12/hunt-study-shows-thyroid-prevents-heart-attacks-by-jeffrey-dach-md.aspx
Links and References:
http://archinte.ama-assn.org/cgi/content/abstract/168/8/855
Arch Intern Med. 2008;168(8):855-860. Thyrotropin Levels and Risk of Fatal Coronary Heart Disease,
The HUNT Study

Arch Intern Med. 2008;168(8):855-860. Background  Recent studies suggest that relatively low thyroid function within the clinical reference range is positively associated with risk factors for coronary heart disease (CHD), but the association with CHD mortality is not resolved.
Methods  In a Norwegian population-based cohort study, we prospectively studied the association between thyrotropin levels and fatal CHD in 17 311 women and 8002 men without known thyroid or cardiovascular disease or diabetes mellitus at baseline.
Results  During median follow-up of 8.3 years, 228 women and 182 men died of CHD. Of these, 192 women and 164 men had thyrotropin levels within the clinical reference range of 0.50 to 3.5 mIU/L. Overall, thyrotropin levels within the reference range were positively associated with CHD mortality (P for trend = .01); the trend was statistically significant in women (P for trend = .005) but not in men. Compared with women in the lower part of the reference range (thyrotropin level, 0.50-1.4 mIU/L), the hazard ratios for coronary death were 1.41 (95% confidence interval [CI], 1.02-1.96) and 1.69 (95% CI, 1.14-2.52) for women in the intermediate (thyrotropin level, 1.5-2.4 mIU/L) and higher (thyrotropin level, 2.5-3.5 mIU/L) categories, respectively.
Conclusions  Thyrotropin levels within the reference range were positively and linearly associated with CHD mortality in women. The results indicate that relatively low but clinically normal thyroid function may increase the risk of fatal CHD.
Author Affiliations: Department of Public Health, Faculty of Medicine (Drs Åsvold and Vatten), and Human Movement Science Programme (Dr Nilsen), Norwegian University of Science and Technology, Trondheim, Norway; St Olavs Hospital, Trondheim University Hospital, Trondheim (Dr Åsvold); Department of Medical Biochemistry, Rikshospitalet-Radiumhospitalet Medical Center, Oslo, Norway (Dr Bjøro); and Department of Social Medicine, University of Bristol, Bristol, England (Dr Gunnell).
http://www.eje-online.org/cgi/content/full/156/2/181
European Journal of Endocrinology, Vol 156, Issue 2, 181-186, 2007  CLINICAL STUDY  The association between TSH within the reference range and serum lipid concentrations in a population-based study. The HUNT Study
Bjørn O Åsvold1,2, Lars J Vatten1, Tom I L Nilsen1 and Trine Bjøro3  1 Department of Public Health, Faculty of Medicine, Norwegian University of Science and Technology, N-7489 Trondheim, Norway, 2 St Olavs Hospital, Trondheim University Hospital, Trondheim, Norway and 3 Department of Medical Biochemistry, Rikshospitalet-Radiumhospitalet Medical Center, Oslo, Norway (Correspondence should be addressed to L J Vatten
Jacob Teitelbaum MD
http://www.endfatigue.com/health_articles_f-n/Heart-low_thyroid_major_cause_heart_attacks.html
Jacob Teitelbaum, MD. Low Thyroid (Even if Tests are Normal) is a Major Cause of Heart Attacks,
William Davis MD
http://heartscanblog.blogspot.com/2008/06/is-normal-tsh-too-high.html
William Davis MD, begin quote:”Is normal TSH too high? There’s no doubt that low thyroid function results in fatigue, weight gain, hair loss, along with rises in LDL cholesterol and other fractions of lipids. It can also result in increasing Lp(a), diabetes, and accelerated heart disease, even heart failure.  But how do we distinguish “normal” thryoid function from “low” thyroid function? This has proven a surprisingly knotty question that has generated a great deal of controversy.  Thyroid stimulating hormone, or TSH, is now the most commonly used index of the adequacy of thyroid gland function, having replaced a number of older measures. TSH is a pituitary gland hormone that goes up when the pituitary senses insufficient thyroid hormone, and a compensatory increase of thyroid hormone is triggered; if the pituitary senses adequate or excessive thyroid hormone, it is triggered to decrease release of TSH. Thus, TSH participates in a so-called “negative feedback loop:” If the thyroid is active, pituitary TSH is suppressed; if thyroid activity is low, pituitary TSH increases.  An active source of debate over the past 10 years has been what a normal TSH level is. In clinical practice, a TSH in the range of 0.4-5.0 mIU/L is considered normal. (Lower TSH is hyperthyroidism, or overactive thyroid; high TSH is hypothyroidism, or underactive thyroid.)  The data from a very fascinating and substantial observation called the HUNT Study, however, is likely to change these commonly-held thyroid “rules.” endquote WIlliam Davis MD
International Hormone Societyhttp://www.intlhormonesociety.org/ref_cons/
Ref_cons_9_thryoid_treatment_of_clinically_hypothyroid_biochemically_hypothyroid_patients.pdf

International Hormone Society, references, clinically hypothyroid , lab euthyrroid
Other Blogs:
http://thyroid.blogspot.com/
Thyroid Blog by Dr. Richard B. Guttler M.D., F.A.C.E.
Location: Santa Monica, California, United States

http://www.stopthethyroidmadness.com/
Stop the Thyroid Madness Blog – a patient tries in vain to convince soctors to switch from Synthroid to Armour
http://thyroid.about.com/
MAry Shomon thyroid blog
Disclaimer click here: http://www.drdach.com/wst_page20.html
The reader is advised to discuss the comments on these pages with his/her personal physicians and to only act upon the advice of his/her personal physician Also note that concerning an answer which appears as an electronically posted question, I am NOT creating a physician — patient relationship.  Although identities will remain confidential as much as possible, as I can not control the media, I can not take responsibility for any breaches of confidentiality that may occur.  Finally, the material produced by myself may be reproduced for personal use, provided that appropriate credit is given
Link to this article:http://wp.me/P3gFbV-LU
http://jeffreydach.com/2008/10/12/hunt-study-shows-thyroid-prevents-heart-attacks-by-jeffrey-dach-md.aspx
(c) 2007-2013 Jeffrey Dach MD All Rights reserved. This article may be copied or reproduced on the internet provided a link and credit is given.
 

Tuesday, June 18, 2013

Are statins and omega-3s incompatible? - Davis

Are statins and omega-3s incompatible?
Posted on June 18, 2013 by Dr. Davis

French researcher, Dr. Michel de Lorgeril, has been in the forefront of thinking and research into nutritional issues, including the Mediterranean Diet, the French Paradox, and the role of fat intake in cardiovascular health. In a recent review entitled Recent findings on the health effects of omega-3 fatty acids and statins, and their interactions: do statins inhibit omega-3?, he explores the question of whether statin drugs are, in effect, incompatible with omega-3 fatty acids.

Dr. Lorgeril makes several arguments:

1) Earlier studies, such as GISSI-Prevenzione, demonstrated reduction in cardiovascular events with omega-3 fatty acid supplementation, consistent with the biological and physiological benefits observed in animals, experimental preparations, and epidemiologic observations in free-living populations.

 2) More recent studies (and meta-analyses) examining the effects of omega-3 fatty acids have failed to demonstrate cardiovascular benefit showing, at most, non-significant trends towards benefit.

He points out that the more recent studies were conducted post-GISSI and after agencies like the American Heart Association’s advised people to consume more fish, which prompted broad increases in omega-3 intake. The populations studied therefore had increased intake of omega-3 fatty acids at the start of the studies, verified by higher levels of omega-3 RBC levels in participants.

In addition, he raises the provocative idea that the benefits of omega-3 fatty acids appear to be confined to those not taking statin agents, as suggested, for instance, in the Alpha Omega Trial. He speculates that the potential for statins to ablate the benefits of omega-3s (and vice versa) might be based on several phenomena:

 –Statins increase arachidonic acid content of cell membranes, a potentially inflammatory omega-6 fatty acid that competes with omega-3 fatty acids. (Insulin provocation and greater linoleic acid/omega-6 oils do likewise.)

–Statins induce impaired mitochondrial function, while omega-3s improve mitochondrial function. (Impaired mitochondrial function is evidenced, for instance, by reduced coenzyme Q10 levels, with partial relief from muscle weakness and discomfort by supplementing coenzyme Q10.)

–Statins commonly provoke muscle weakness and discomfort which can, in turn, lead to reduced levels of physical activity and increased resistance to insulin. (Thus the recently reported increases in diabetes with statin drug use.)

Are the physiologic effects of omega-3 fatty acids, present and necessary for health, at odds with the non-physiologic effects of statin drugs?

I fear we don’t have sufficient data to come to firm conclusions yet, but my perception is that the case against statins is building. Yes, they have benefits in specific subsets of people (none in others), but the notion that everybody needs a statin drug is, I believe, not only dead wrong, but may have effects that are distinctly negative. And I believe that the arguments in favor of omega-3 fatty acid supplementation, EPA and DHA (and perhaps DPA), make better sense.

 - See more at: http://blog.trackyourplaque.com/2013/06/are-statins-and-omega-3s-incompatible.html

Wednesday, August 8, 2012

I Wish I Had Lipoprotein(a)!

I Wish I Had Lipoprotein(a)!


Why would I say such a thing? Well, a number of reasons. People with lipoprotein(a), or Lp(a), are, with only occasional exceptions:

Very intelligent. I know many people with this genetic pattern with IQs of 130, 140, even 160+.

Good at math–This is true more for the male expression of the pattern, only occasionally female. It means that men with Lp(a) gravitate towards careers in math, accounting, financial analysis, physics, and engineering.

Athletic–Many are marathon runners, triathletes, long-distance bicyclists, and other endurance athletes. I tell my patients that, if they want to meet other people with Lp(a), go to a triathlon.

Poor at hydrating. People with Lp(a) have a defective thirst mechanism and often go for many hours without drinking water. This is why many Lp(a) people experience the pain of kidney stones: Prolonged and repeated dehydration causes crystals to form in the kidneys, leading to stone formation over time.

Tolerant to dehydration–Related to the previous item, people with Lp(a) can go for extended periods without even thinking about water.

Tolerant to periods of food deprivation or starvation–More so than other people, those with Lp(a) are uncommonly tolerant to days without food, as would occur in a wild setting.

In short, people with Lp(a) are intelligent, athletic, with many other favorable characteristics that provide a survival advantage . . . in a primitive world.

So when did Lp(a) become a problem? When an individual with Lp(a) is exposed to carbohydrates, especially those from grains. When an evolutionarily-advantaged Lp(a) individual is exposed to carbohydrates, more than other people they develop:

–Excess quantities of small LDL particles–Recall that Lp(a) is a two-part molecule. One part: an apo(a) made by the liver. 2nd part: an LDL particle. When the LDL particle within the Lp(a) molecule is small, its overall behavior is worse or more atherogenic (plaque-causing).

–Hyperglycemia/hyperinsulinemia–which then leads to diabetes. Unlike non-Lp(a) people, these phenomena can develop with far less visceral fat. A Lp(a) male, for instance, standing 5 ft 10 inches tall and weighing 150 pounds, can have as much insulin resistance/hyperglycemia as a non-Lp(a) male of similar height weighing 50+ pounds more.

Key to gaining control over Lp(a) is strict carbohydrate limitation. Another way to look at this is to say that Lp(a) people do best with unlimited fat and protein intake.
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Read the complete article here.

Sunday, July 1, 2012

Dr Davis was interviewed by   recently. The full interview can be seen here. I have shown only the part that specifically addresses heart disease.
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2. How is wheat consumption linked to heart health?

Wheat consumption causes heart disease. It’s not cholesterol, it’s not saturated fat that’s behind the number one killer of Americans; it’s wheat.

The nutrition community has been guilty of following a flawed sequence of logic: If something bad for you (white processed flour) is replaced by something less bad (whole grains) and there is an apparent health benefit, then a whole bunch of the less bad thing is good for you. Let’s apply that to another situation: If something bad for you—unfiltered Camel cigarettes—are replaced by something less bad—filtered Salem Cigarettes—then the conclusion would be to smoke a lot of Salems. The next logical question should have been: What is the health consequence of complete removal? Only then can you observe the effect of whole grains vs. no grains . . . and, from what I witness every day, you see complete transformations in health.

Consumption of wheat, due to its unique carbohydrate, amylopectin A, triggers formation of small, dense LDL particles more than any other common food. Small, dense LDL particles are the number one cause for heart disease in the U.S. The majority of adults now have an abundance of small LDL particles because they’ve been told to cut their fat and “eat plenty of healthy whole grains.” This situation of excessive small LDL particles can appear on a conventional cholesterol panel as higher levels of LDL (“bad”) cholesterol, along with low HDL cholesterol and higher triglycerides that often leads to statin drugs. When more sophisticated lipoprotein testing is obtained, then the explosion of small LDL particles becomes obvious.

Compound this with the increased appetite triggered by the gliadin protein in wheat that acts as an appetite-stimulant, and you gain weight. The weight gained is usually in the abdomen, in the deep visceral fat that triggers inflammation, what I call a “wheat belly.” Wheat belly visceral fat is a hotbed of inflammation, sending out inflammatory signals into the bloodstream and results in higher blood sugar, blood pressure, and triglycerides, all adding up to increased risk for heart disease.
Say goodbye to wheat and small LDL particles plummet, followed by weight loss from the wheat belly visceral fat. Inflammation subsides, blood sugar drops, blood pressure drops. In short, elimination of wheat is among the most powerful means of reducing risk for heart disease.

Wednesday, March 7, 2012

Massaging the truth


Massaging the truth

Massage therapist, Jonathan Sugai, posted this interesting comment on Amazon:

I first learned about Wheat Belly back in September 2011 in Northern California. My friend Seamus was eating some chili out of a can while we were waiting for our class at a seminar to start. I asked him why he was doing that and he said, “You gotta read this book called Wheat Belly!”

After he explained what the book was about, how quickly and dramatically his situation improved (his digestion turned around very quickly!), it made total sense why he was eating chili out of a can even though we had plenty of food served (most of it had some form of wheat in it!).

I immediately downloaded the book off Amazon and finished reading it on my way back home to Hawaii and my view on food had totally shifted. Life would never be the same again!

I knew eating bread would contribute to weight gain, however learning about all the OTHER information about the hybridization, the Gliadin, and all the blood sugar, inflammation, digestive, and neurological effects, OMG!!!

I am a massage therapist and part of a family business and now it is clear why we have so many people coming in with so many aches, pains, & injuries from seemingly routine activities. It is clear to me that consuming a diet with wheat triggers inflammation and creates all sorts of systemic problems and their bodies are no longer able to tolerate typical activities.

I realize why I’m seeing more clients not responding to our techniques that used to work well. The techniques are great, however the clients that are coming in are unhealthier than ever with rampant inflammation, pre-diabetic / diabetic, high cholesterol, nervous systems interfered with by gliadin, overweight, etc. etc. etc.

All of our clients who have made the transition to wheat free have lost weight, have more energy, and most of the joint pains and stiffness have gone away and their massage treatments are enjoyable again and we aren’t putting out fires anymore!

It is our responsibility to share and let everyone know in order to create change. Our friends & family will continue to see their health degrade if they continue on this path.


Until reading his comment, it hadn’t occurred to me that massage therapists could provide some very interesting observations on wheat elimination. I find it fascinating that, once his clients said goodbye to wheat, they responded better to his efforts.

Any other massage therapists out there that would like to weigh in?


http://www.wheatbellyblog.com/2012/03/massaging-the-truth/



Saturday, March 3, 2012

The followers of the Track Your Plaque program enjoy virtual elimination of risk.

Why are heart attacks still happening?

I’m a cardiologist. I see patients with heart disease in the form of coronary artery disease every day.
These are people who have undergone bypass surgery, received one or more stents or undergone other forms of angioplasty, have survived heart attacks or sudden cardiac death, or have high heart scan scores. In short, I see patients every day who are at high-risk for heart attack and death from heart disease.

But I see virtually no heart attacks. And nobody is dying from heart disease. (I’m referring to the people who follow the strategies I advocate, not the guy who thinks that smoking a pack of cigarettes a day is still okay, or the woman who thinks the diet is unnecessary because she’s slender.)
Two high-profile deaths from heart attacks occurred this week:

Davy Jones–The iconic singer from the 1960s pop group, the Monkees, suffered sudden cardiac death after a large heart attack, just hours after experiencing chest pain.

Andrew Breitbart–The conservative blogger and controversy-generating media personality suffered what was believed to be sudden cardiac death while walking.

It’s a darn shame and it shouldn’t happen. The tools to identify the potential for heart attack are available, inexpensive, and simple. The strategies to reduce, even eliminate, risk are likewise available, inexpensive, and cultivate overall health.

The followers of the Track Your Plaque program who
1) get a heart scan that yields a coronary calcium score (for long-term tracking purposes)
2) identify the causes such as small LDL particles, lipoprotein(a), vitamin D deficiency, and thyroid dysfunction
3) correct the causes
enjoy virtual elimination of risk.
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http://www.trackyourplaque.com/blog/2012/03/why-are-heart-attacks-still-happening.html

Friday, February 24, 2012


The Wheat Belly Diet

The cardiologist-created Wheat Belly Diet is built on the premise that wheat, not sweets, is making you fat. Here's how a wheat-free diet may help you lose weight.

Forget your beer belly — William Davis, MD, a preventive cardiologist in Milwaukee, Wisc., says your wheat belly is the real health hazard. Davis’ prescription for a whittled middle is simple: Cut all wheat from your diet. Better yet, Davis argues in his book, Wheat Belly: Lose the Wheat, Lose the Weight, and Find Your Path Back to Health, that eating wheat-free will both prevent and reverse health problems such as acne, cataracts, diabetes, heart disease, and arthritis.

The Wheat Belly Diet suggests we get back to eating more like our ancestors who existed solely on foods found in nature, not those grown for production or manufactured for sale. In that way, the diet is similar to another popular diet, the Paleo or hunter-gatherer diet, says Joan Salge Blake, MS, RD, a Boston nutritionist, author of Nutrition & You: Core Concepts for Good Health, and a spokeswoman for the American Dietetic Association. Here’s how to find out if going wheat-free is right for you.

The Wheat Belly Diet: What Is It?

Your menu choices on this eating plan include natural foods such as eggs, nuts, vegetables, fish, poultry, and other meats. You can use herbs and spices freely and healthy oils, such as olive and walnut, liberally. Eat fruit occasionally — just one or two pieces a week — because the naturally occurring fructose in fruit is a simple carbohydrate. As part of this diet, you’re required to eliminate all fast food, processed snacks, and junk foods, and drink lots of water.

The Wheat Belly Diet is in fact gluten-free, but Davis doesn’t advocate eating packaged gluten-free foods. His reasoning: These products often simply substitute brown rice, potato starch, rice starch, tapioca starch, or cornstarch for wheat flour, and those substitutes can raise your blood sugar or glucose higher than wheat.

The Wheat Belly Diet: How Does It Work?

Cut wheat from your diet, and you’ll eat about 400 fewer calories a day than you normally would, Davis says. This calorie deficit alone is almost enough to add up to a pound of weight loss per week. “Anything that is going to cut calories is going to work because losing weight is a numbers game,” Blake says. “Eat fewer calories than you burn, and you’ll lose weight. Likewise, eat more than you burn, and you’ll gain weight.”Another reason the diet works, Davis says, is that wheat contains a unique protein, gliadin, which stimulates your appetite— so when you eat wheat, your body just wants more wheat. Eliminate wheat and your appetite diminishes on its own. Wheat also causes blood sugar spikes, and elevated blood-sugar levels can cause your body to store calories as fat. Lower your blood sugar by eliminating wheat, and it can contribute to weight loss.

The Wheat Belly Diet: Sample Menu

Breakfast: Plain yogurt with berries and almonds
Lunch: Grilled chicken breast with salsa, 1/2 cup brown rice, steamed vegetables sprinkled with extra-virgin olive oil
Dinner: Baked eggplant topped with mozzarella cheese and tomato sauce, mixed green salad spritzed with extra-virgin olive oil
Snacks: Black-bean dip and raw vegetables

The Wheat Belly Diet: Pros


  • If you adhere strictly to the diet, you will lose weight. Over three to six months, you can lose 25 to 30 pounds depending on your age, gender, and physical activity, Davis says.
  • The diet is simple. There’s no need to count calories, limit portions, or calculate fat grams. All you have to do is eliminate foods that contain wheat.
  • The diet is rich in vegetables, which are full of vitamins and fiber. Eating a diet rich in fruits and vegetables can help lower cholesterol, stabilize blood sugar, and reduce the inflammation that can cause conditions from acne to arthritis.

The Wheat Belly Diet: Cons


  • The diet is restrictive, and it may be hard to maintain for the long-term, especially if foods such as bread, cookies, and pasta are among your favorites. “Losing weight doesn’t have to be this challenging,” Blake says. “Do you really need to go to this extreme?”
  • Wheat is in a huge number of packaged foods. You have to read food labels carefully because it can be hidden in everything from chewing gum to granola as an emulsifier or leavening agent.
  • When you remove all wheat from your diet, if you “cheat” and eat a slice of whole-wheat toast or half a bagel, the wheat could cause digestive problems, such as stomach cramps and gas.
  • You could be missing out on some important nutrients. “Whenever you limit whole types of foods, you have to make sure you’re eating healthfully,” Blake says. “This isn’t a well-balanced diet. You should sit down with a registered dietitian to be sure you’re meeting all your nutrient needs if you choose this diet.”
  • Although you can lose weight with this diet, it will be lost from all over your body, not just your “wheat belly” or love handles, Blake says. Weight loss doesn’t work that way — you don’t lose from a specific area.

The Wheat Belly Diet: Short-Term and Long-Term Effects

The foods you can eat on the Wheat Belly Diet are healthy, and you should lose weight rapidly if you stick to the plan. Weight loss can affect more than just your appearance: Study after study has shown it can boost heart health, reduce pain, improve your energy levels, and more. For example, someone who is prediabetic and loses just 15 pounds can reduce the risk for diabetes over three years by 58 percent, Blake says.

Because the diet is so new, not much is known about the long-term effects, Blake says, but serious health consequences are not anticipated. Overall, Blake remains skeptical.

“There’s nothing wrong with wheat,” she says.“It isn’t wheat that’s causing you to gain weight; it’s the calories you’re eating. Just eat more fruits and vegetables as part of a balanced diet, and you can cut calories and lose weight while still occasionally eating foods that contain wheat.”
Last Updated: 02/23/2012
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Wednesday, February 22, 2012

A Headline You Will Never See: 60 Year Old Man Dies of Cholesterol

Cholesterol scam: Disinformation slowly unraveling among health professionals

Learn more: http://www.naturalnews.com/035033_cholesterol_disinformation_fats.html#ixzz1n7iFu2ir

(NaturalNews) The idea of cholesterol creating cardiac problems has caused obsessive cholesterol count blood testing for decades. Another outcome of this scare was obsessively avoiding fat, especially saturated fats.

The food industry responded with low and no fat foods from milk to cottage cheese and more. Processed foods promoted their low or no fat contents as though they were the healthiest foods in the freezer.

Healthy fats such as coconut oil and palm oil were spurned and replaced by very unhealthy trans-fat, processed and heated cooking oils. Relatively healthy whole butters were replaced by plastic margarines.

However, this myth of cholesterol dangers lurking in saturated fats waiting to clog your arteries and cause you to die of cardiac arrest is beginning to unravel.

Unraveling the myth of cholesterol

A meta-analysis of properly performed previous studies on heart health and saturated fats concluded there was no association between cardiac issues and saturated fats. This was published in the American Journal of Clinical Nutrition (AJCN) on January 13th, 2010. (1)

Meta-analysis is a statistical method of proving or disproving varied epidemiological studies within a set topic. The AJCN meta-analysis covered studies involving 350,000 subjects who were followed for 5 to 23 years.

The trend set by the saturated fat high cholesterol disinformation a few decades ago has resulted in many Americans eating less fat and showing lower blood cholesterol levels. Yet, heart disease rates have continued to rise along with diabetes, pre-diabetes and obesity. (1)

Dr. William Davis explains in his article "A Headline You Will Never See: 60 Year Old Man Dies of Cholesterol" that cholesterol doesn't kill "any more than a bad paint job on your car could cause a fatal car accident." (1)

He explains the cause of most heart attacks and coronary problems is atherosclerotic plaque in the coronary arteries, which can build up and rupture or clog the arteries. He goes on to describe other factors that can cause plaque ruptures, including inflammatory pneumonia.

Though there can be some cholesterol in the plaque, cholesterol itself is waxy and pliable. Cholesterol is important for brain cells, nerves and other cellular structural components. Calcium deposits (calcification) in artery interiors are much worse components of plaque. It belongs in your bones and not in your arteries. Vitamin K2 helps transport calcium out of your blood and into your bones.

Dr. Davis recommends avoiding cholesterol panels for heart health concerns and opting for a measure of coronary atherosclerotic plaque.

The scam continues despite overwhelming contradictory evidence

Despite more and more published journals and doctors proving coronary heart disease (CHD) is not caused by high saturated fat diets and cholesterol, the myth persists. Many peoplewith low cholesterol have died of CHD while in their 40s, while many with high cholesterol never have CHD issues.

Several studies of heart attack cadavers have also revealed the disinformation of cholesterol dangers. Yet the common advice from cardiologists upon seeing high cholesterol is to get an angiogram,adiagnostic testwhichis expensive and not so safe. Then there are those pricey drugs meantto lower cholesterol while wreaking havoc on overall health. (2)

Cholesterol is vital for many functions. For example, it helps convert sunlight into vitamin D3. If you're not getting enough with your food, the liver is forced to manufacture it. Low cholesterol has been linked to higher stroke risks.

Oxidized cholesterol from hydrogenated and refined polyunsaturated cooking oils and margarine can lead to complications that result in CHD. This comes not only directly from the oils themselves, but indirectly from the oxidation process those oils initiate. (3)

These toxic oils and butter substitutes were created to replace thewholesome saturated fats that should be consumed.

Sources for this article include:

(1)
http://www.sott.net

(2) http://www.opednews.com

(3) http://www.treelight.com/health/healing/Cholesterol.html

About the author:
Paul Fassa is dedicated to warning others about the current corruption of food and medicine and guiding others toward a direction for better health with no restrictions on health freedom. You can visit his blog at
http://healthmaven.blogspot.com


Learn more: http://www.naturalnews.com/035033_cholesterol_disinformation_fats.html#ixzz1n7i8Tdlx

Read the article here > http://www.naturalnews.com/035033_cholesterol_disinformation_fats.html

Wednesday, February 8, 2012

Wheat and Heart Disease (Ch 10)

"So it's not really about cholesterol. It's about the particles that cause atherosclerosis. Today you and I are able to directly quantify and characterize lipoproteins, relagating cholesterol to join frontal lobotomies in the outdated medical practice garbage dump in the sky."

Wheat Belly - William Davis, MD  (p 148)
www.rodalebooks.com

Monday, February 6, 2012

60-Year Old Man Dies of High Cholesterol

60-year old man dies of high cholesterol

Never saw a headline like this? Neither have I. That’s because it doesn’t happen.

Cholesterol doesn’t harm, maim, or kill. It is simply used as a crude–very crude–marker. It is, in reality, a component of the body, of the cell wall, of lipoproteins (lipid-carrying proteins) in the bloodstream. It is used a an indirect gauge, a “dipstick,” for lipoproteins in the blood to those who don’t understand how to identify, characterize, and quantify actual lipoproteins in the blood.

Cholesterol itself never killed anybody, any more than a bad paint job on your car could cause a fatal car accident.

What kills people is rupture of atherosclerotic plaque in the coronary arteries. For all practical purposes, you must have atherosclerotic plaque in order for it to rupture (much like a volcano erupts and spews lava). It’s not about cholesterol; it’s about atherosclerotic plaque. Plaque might contain cholesterol, but cholesterol is not the thing itself that causes heart attack and death.

So why do most people obsess about cholesterol? Good question. It is, at best, a statistical marker for the possibility of having atherosclerotic plaque that ruptures. High cholesterol = higher risk for heart attack, low cholesterol = lower risk for heart attack. But the association is weak and flawed, such that people with high cholesterol can live a lifetime without heart attack, people with low cholesterol can die at age 43.The same holds true for LDL cholesterol, you know, the calculated value based on flawed assumptions about LDL’s relationship to total cholesterol, HDL cholesterol, and VLDL cholesterol.

A crucial oversight in the world of cholesterol: There are many other factors that cause atherosclerotic plaque and its rupture, such as inflammatory phenomena, calcium deposition, artery spasm, hemorrhage within the plaque itself, degradative enzymes, etc., none of which are suggested by cholesterol measures.

But one observation has held up, time and again, over the past 40 years of observations on coronary disease: The greater the quantity of coronary atherosclerotic plaque, the greater the risk of atherosclerotic plaque rupture. An increasing burden of atherosclerotic plaque along the limited confines of coronary arteries, just a few millimeters in diameter and a few centimeters in length, is like a house of cards: It’s bound to topple sooner or later, and the bigger it gets, the less stable it becomes.

If you are concerned about future potential for heart disease and heart attack, don’t get a cholesterol panel. Get a measure of coronary atherosclerotic plaque.

Sunday, January 29, 2012

Back to basics: Coronary calcium


Back to basics: Coronary calcium

After having my attentions pulled a thousand different directions these past 6 months, with the release of Wheat Belly and all the wonderful media attention it has attracted, I’ve decided to pick up here with a series of discussions about the fundamental issues important to the Track Your Plaque program and prevention and reversal of coronary atherosclerotic plaque.

I fear the discussions at times have drifted off into the exotic. This is great because this is how we learn new lessons, but we can never lose sight of the basics, else we risk losing control over this disease.

Imagine you’ve got a beautiful new car. You wax it, gap the spark plugs, rotate the tires, etc. and it looks brand-new, just like it came off the dealer’s lot. 50,000 miles pass, however, and you realize you’ve forgotten to change the oil. Ooops! In other words, no matter how meticulous the attention to transmission, tires, and paint job, neglect of the most basic responsibility can ruin the whole thing. We can’t let that happen with heart health.

If we propose to reverse coronary atherosclerotic plaque, we’ve got to have something to measure. First, it tells us whether we have atherosclerotic plaque in the first place, the stuff that accumulates and blocks flow and causes anginal chest pains, and ruptures like a little volcano and causes heart attacks. Second, it gives us something to track over the years to know whether plaque has grown, stopped growing, or been reduced. Without such a measure, you will be driving without a speedometer or odometer, just guessing whether or not you’ve gotten to your destination.

Of course, the conventional approach to heart disease and heart attack is not to track atherosclerotic plaque in your coronary arteries, but to track some distant “risk factor” for atherosclerotic plaque, especially LDL cholesterol. But LDL cholesterol is flawed at several levels. First, it is calculated, not measured. The nearly 50-year old Friedewald equation used to calculate LDL cholesterol is based on several flawed assumptions, yielding a value that can be 20, 30, or 50% inaccurate as a rule, only occasionally generating a value close to the real value. (No point in publicizing this problem, of course: Why compromise a $27 billion annual cash cow?) It also ignores the effect of diet. (No, cutting fat does not reduce LDL for real, only the calculated value. Cutting carbohydrates, especially wheat–”healthy whole grains”–slashes measured LDL values like NMR LDL particle number and apoprotein B.)

But all risk factors are, at best, snapshots of the situation at that moment in time. They change from day to day, week to week, month to month, year to year. If you do something dramatic in health, like lose 50 pounds, you can substantially change your risk factors values, like LDL cholesterol and HDL cholesterol. But you may not modify the amount of atherosclerotic plaque in your heart’s arteries.

Measuring the amount of atherosclerotic plaque in your heart’s arteries is, in effect, a cumulative expression of the effects of risk factors up until the moment of measurement.

There are several stumbling blocks, however, in the concept of measuring coronary atherosclerotic plaque. We cannot measure all the unique components of plaque, such as fibrous tissue like collagen, or degradative enzymes like collagenases, or inflammatory proteins like matrix metalloproteinase, or the debris of hemorrhage and inflammation. We struggle to contemporaneously mix in measures of bloodborne inflammation, coagulation and viscosity, and physiological phenomena of the artery itself, like endothelial dysfunction, medial (muscle) tone, and adventitial fat.

So we are left with semi-static measures of total coronary atherosclerotic plaque like coronary calcium, obtainable via CT heart scans as a calcium “score.” No, it is not perfect. It does not reflect that moment’s blood viscosity, it does not reflect the inflammatory status of the one nasty plaque in the mid-left anterior descending, nor does it reflect the irritating sheer effects of a blood pressure of 150/95.

But it’s the best we’ve got.

If anyone has something better, I invite you to speak up. Carotid ultrasound, c-reactive protein, ankle-brachial index, stress nuclear studies, myoglobin, skin cholesterol, KIF6 genotype . . . none of them approach the value, the insight, the trackability of actually measuring coronary atherosclerotic plaque. And the only method we’ve got to gauge coronary atherosclerotic plaque that is non-invasive and available in 2012? Yup, a good old CT heart scan calcium score.


http://www.trackyourplaque.com/blog/2012/01/back-to-basics-coronary-calcium.html?utm_source=feedburner&utm_medium=feed&utm_campaign=Feed%3A+blogspot%2Ftpzx+%28The+Heart+Scan+Blog%29&utm_content=Google+Reader

Sunday, October 9, 2011

Why small LDL particles are the #1 cause of heart disease in the US



Why small LDL particles are the #1 cause of heart disease in the US

Posted on by Dr. William Davis

Ask your doctor: What is the #1 cause of heart disease in the US?

Let’s put aside smoking, since it is an eminently modifiable risk and none of those crazies read this blog anyway. What will your doctor say? Most like he or she will respond:

High cholesterol or high LDL cholesterol
Too much saturated fat
Obesity

Pfizer, Merck, AstraZeneca and their kind would be overjoyed to know that they can add your doctor to their eager following.

I’d tell you something different. I would tell you that small LDL particles are, by far and away, the #1 cause for heart disease. I base this claim on several observations:

–Having run over 10,000 lipoprotein panels (mostly NMR) over the past 15 years, it is a rare person who does not have a moderate, if not severe, excess of small LDL particles. 50%, 70%, even 90% or more small LDL particles are not rare. Over the course of a year, the only people who show no small LDL particles are slender, athletic, pre-menopausal females.

–In studies in which lipoproteins have been quantified in people with coronary disease, small LDL particles dominate, just as they do in my office. Here’s a 2006 review.

–Small LDL is largely the province of people who consume carbohydrates, such as the American population instructed to “cut fat and eat more healthy whole grains.” Conventional diet advice has therefore triggered an expllosion in small LDL particles.

–When fasting triglycerides exceed 60 mg/dl, small LDL particles increase as a proportion of total LDL particles. This includes the majority of the US population. (This ignores postprandial, or after-eating, triglycerides, which also contribute to small LDL formation.)

If you were to read the data, however, you might conclude that small LDL affects a minority of people. This is because in most studies small LDL categorize it as either “pattern B,” meaning exceeding some arbitrary threshold of percentage of small LDL particles, versus “pattern A,” meaning falling below that same arbitrary threshold.

Problem: There is no consensus on what percentage of small LDL particles should mark the cutoff between pattern A vs. pattern B. In many studies, for instance, people with 50% small LDL particles are called “pattern A.”

If, instead, we were to set the bar lower to identify this highly atherogenic (atherosclerotic plaque-causing) particle at, say, 20-30% of total, then the number or percentage of people with “pattern B” small LDL particles would go much higher.

I see this play out in my office and in the online program, Track Your Plaque, every day: At the start eating a low-fat, grain-filled diet with lots of visceral fat (“wheat belly”) to start, they add back fat and cut out all wheat and limit carbohydrates. Small LDL particles plummet
 

About Dr. William Davis

Dr. Davis is Medical Director of the Track Your Plaque program and advocate of early heart disease prevention and reversal. He practices preventive cardiology in Milwaukee, Wisconsin.

Saturday, October 1, 2011

The David Korech of Medicine?

Tom Naughton's critical review of a critical review that must have been so off base, mis-leading, false etc. that the powers that be have removed it from the shelves.

=====================================================

The Long Knives Are Out For ‘Wheat Belly’

from Fat Head
Look as if the grain-industry people have been trolling the internet attempting to trash Wheat Belly, the outstanding new book by Dr. William Davis. How else do you explain an Amazon review written under the title The David Korech of Medicine. Notice that the review doesn’t dispute any of the facts or science presented in Wheat Belly:
The author has no credentials, no credibility, just a small cult of terriby misinformed followers. Don’t be fooled by the high volume screech against wheat and grains.
I have to admit, it didn’t occur to me when I recommended Wheat Belly that Dr. Davis has no credentials. I assumed being a doctor who’s seen the benefits of a wheat-free diet in thousands of his own patients counted for something. I also figured that after poring over hundreds of studies on wheat’s health effects and thoroughly researching the history and genetic structure of wheat, Dr. Davis was qualified to write on the topic.
Allegations of “secret ingredients in wheat” to make you eat more, or comparisons to cigerettes. Seriously?!
Apparently the wheat lobby doesn’t require its job applicants to pass a spelling test. Are cigerettes similar to cigarettes?
Dr. Davis never claimed there are secret ingredients in wheat. He didn’t (as far as I know) don a Ninja costume or disguise himself as a plumber and break into the headquarters of Wheat , Inc. to pilfer documents. He simply described, based on published documents, how wheat has been modified during the past 50 years.
For over 8000 years wheat has sustained and grown human kind …
Eight-thousand years ago, humans (some of them, anyway) consumed Einkorn wheat. The main similarity between Einkorn wheat and today’s mutant wheat is they’re both called “wheat.” We also call both Thomas Jefferson and Adolph Hitler “humans,” but they produced rather different effects on other humans. What we ate 8,000 years ago has nothing to with the health consequences of eating a highly modified variety of wheat that’s only been around for the past 50 years.
And wheat didn’t “grow” humankind. Wheat shrunk humankind.

… oh and it tastes good when mixed with a little water and yeast.
That explains why nearly every brand of bread includes high fructose corn syrup high on the list of ingredients.
Every nutritionist and serious medical professional will tell you that bread is the most economical and safe source of essential nutrients.

Every one, seriously? That’s odd … I seem to recall a lot of conversations and interviews with nutritionists and medical professionals who agree wholeheartedly with Dr. Davis — who, despite having a good sense of humor, is a serious medical professional. So serious that he wrote a book about wheat.

In fact, bread is handed out in natural disasters because it sustains life without food safety issues or requiring refrigeration.

Tell ya what: if I’m ever starving because a tornado wiped out all the refrigerators within a 50-mile radius, I’ll eat some of the bread FEMA hands out. But the fact that bread doesn’t require refrigeration doesn’t in any way prove it’s good for us. Coca-Cola and heroin don’t require refrigeration either.
And now, suddenly it will kill you. Comical!
No, today’s mutant wheat doesn’t kill you suddenly. It’s more like slow torture.
This book is such a bone headed, misinformed way to just scare people into not eating.
Now wait a minute, Buster! Dr. Davis is 100% in favor of people eating. He just doesn’t want them eating a mutant grain product that will make them fat and sick.
As for secret ingredients, humm, apparently the author is ignorant of the food laws that regulate everything that goes into food and on food labels. Unlike some enforcement agencies, the FDA has some serious teeth behind its enforcement.
Well, that certainly negates all the studies demonstrating the negative effects of wheat that Dr. Davis cited in Wheat Belly. If the USDA and FDA are on the beat, mutant wheat (which is heavily subsidized by the same government that funds the USDA and FDA) simply has to be safe. That’s why they conducted all those tests on the stuff to make sure it wouldn’t cause any … no, wait a minute. Nobody ever conducted tests on mutant wheat to see if it’s fit for human consumption. Okay then, we’ll just have faith the USDA and FDA can tell if a food product is safe by looking at it. That works for me.
As for frankenwheat, again seriously?! Wheat, due to its ubiquitous presence in the world is treated as sacrosant from any GMO research or development.
I see. So when wheat went from being a wispy plant that couldn’t be harvested until it was five feet tall to a short, stubby little plant that’s harvested when it’s two feet tall — all in just the past 50 years – that was a natural mutation, was it? Boy, evolution works really, really fast. I wonder what all those plant geneticists on the Monsanto payroll are doing all day. They’re probably just sitting around, playing poker and thinking, “Geez, I wish they’d let us develop some kind of hybridized new seeds that the company could patent and then force farmers to buy by threatening to sue them if some of those seeds blow onto their farms and start growing even if the farmers didn’t want the seeds in the first place.”
If you need real, science based information on healthy eating, check out [...] and leave this book and its cult in the compound.
I’ll try to read [...], but when I searched for […] on Amazon, all that came up was a kids’ book titled “Dot and Dash play Dot-Dot-Dot.”
I was hoping the grain lobby wouldn’t find out about our cult. For those of you who haven’t been recruited yet, we have a secret handshake that requires a lot of flexibility in the fingers — that keeps out the grain-lobby infiltrators who are suffering from wheat-induced arthritis. At our meetings, Dr. Davis sacrifices a goat and then we all spend hours enjoying ritual dancing around a bonfire on our pain-free legs. Sometimes we even dance naked. It’s not a sexual thing, you understand … we just like showing off our rash-free skins.
I’m sure this campaign by the grain industry will succeed. Any day now, Dr. Davis will have a conversation with a patient something like this:
“So how do feel after being wheat-free for the past two months?”
“Well, Dr. Davis, I lost 25 pounds, my psoriasis cleared up, my arthritis went away, I’m sleeping better, I’m not depressed anymore, I think more clearly, and I stopped wheezing.”
“Excellent! So you’ll continue avoiding wheat?”
“Of course not.”
“But—“
“Wheat has sustaining humans for 8,000 years, Dr. Davis, so it has to be good for us. Besides, I don’t want to end up spending all day the airport banging on a tambourine and selling flowers to passengers.”
“But I never asked you to– why are you sticking your fingers in your ears?”
“What?”
“I said, WHY ARE YOU STICKING YOUR FINGERS IN YOUR EARS?”
“My de-programmers told me not to listen to you.”